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Is a result of a damage of motor fibers traveling from the cerebral cortex to the lower brain stem ?
Is a result of a lower motor neuron lesion of the nerves in the lower brain stem?
Normal emotions?
Labile emotions?
Spastic tongue?
Wasted tongue, fasciculations?

Upper motor neurons (UMN)
Lower motor neurons (LMN)

Pyridostigmine is a reversible acetylcholinesterase inhibitor?
Amifampridine blocks potassium channel efflux in nerve terminals which increases duration of the action potential?
Aminoglycoside antibiotics can aggravate myasthenia gravis (MG) ?
Patients with MG show decreased susceptibility to depolarizing neuromuscular blocking agents ?
Patients with MG show increased susceptibility to non-depolarizing neuromuscular blocking agents?
Patients with Lambert-Eaton myasthenic syndrome show decreased susceptibility to both depolarizing and non-depolarizing neuromuscular blockers?

When action potential arrives at the presynaptic neuron terminal, voltage-dependent calcium channels open and calcium ions flow from the extracellular fluid into the neuron?
Influx of calcium into the neuron triggers the fusion of acetylcholine vesicles with the membrane and the release of ACh into the synaptic cleft?
Nicotinic and muscarinic ACh receptors (AChRs) are ligand-gated ion channels?
The binding of ACh to nicotinic AChR in the postsynaptic muscle membrane opens a calcium channel within the receptor allowing calcium influx and depolarization of the muscle membrane?
Two ACh molecules must bind with each AChR to open its channel?
Choline acetyltransferase in the synaptic cleft decomposes ACh into choline and acetic acid?

The titer of anti-AChR antibodies in patients with myasthenia gravis (MG) closely matches the severity of symptoms?
The sensitivity of anti-AChR antibodies with pure ocular MG is between 85 – 90% ?
Repetitive nerve stimulation typically shows decremental response in MG?
Repetitive nerve stimulation typically shows facilitation in Lambert-Eaton myasthenic syndrome?
Single-fiber electromyography is the most sensitive test for MG?
Subjective improvement in muscular weakness after intravenous edrophonium is considered as positive Tensilon test and confirmation of MG ?

Antibodies against the nicotinic acetylcholine receptor (AChR) are found in 50 – 60% of patients with generalized myasthenia gravis (MG)?
A majority of antibodies against the AChR found in patients with MG are “blocking” antibodies which prevent binding of acetylcholine to the AChR?
Anti-AChR “binding” antibodies, bind to the AChR and cause destruction of junctional folds by activating the complement system?
Most of the seronegative MG patient (those that do not have anti-AChR antibodies but have MG) have antibodies against P/Q-type VGCC (voltage gated-calcium channels)?
Antibodies against LRP4, Agrin and titin proteins are shown in some seronegative MG patients ?
Patients that have anti-MuSK (Muscle-Specific Kinase) antibodies suffer from Lambert–Eaton myasthenic syndrome?

According to “The 2016 World Health Organization Classification of Tumors of the Central Nervous System”: